Physiologic bowling of toddlers |
Femur and tibia are mildly bowed. |
Predilection for the first 2 y of life. Corrected within 6 mo of walking. Reaches adult pattern of mild valgus by 6–7 y. |
Plastic/bowing fracture |
Bowing of radius or ulna are the most common sites. |
If the radius is fractured, the accompanying bone (ulna) may be fractured (and vice versa). |
Rickets
Fig. 5.90 |
(see Table 5.42 ) |
Widening of the medial aspect of the growth plate in older children with an ossified distal femoral epiphysis. The weight-bearing part is always the widest. |
Recurrent fracture with malposition |
Bowing of fragile bones after repeated fractures. |
Occurs in all bone disorders with increased bone fragility (e.g., osteogenesis imperfecta and other intrinsic sclerotic bone dysplasias). |
Fibrous dysplasia
Fig. 5.91a, b, p. 558 |
Expansile and lucent lesion with a ground-glass matrix. |
Typically restricted to the polyostotic variety. Medial bowing of the proximal femur (Shepherd crook deformity). |
Intrauterine malposition/congenital bowing of the tibia |
Unilateral bowing, mostly involving the tibial diaphysis. Lateral and posterior bowing. Fibula is also bowed. |
Believed to result from an abnormal intrauterine position. Usually heals in the neonatal period. |
Neurofibromatosis
Fig. 5.92a, b, p. 558 |
Anterolateral bowing. |
Ten percent of patients with neurofibromatosis will have pseudarthrosis of the tibia. |
Tibia vara (Blount disease)
Fig. 5.43, p. 524 |
Varus angulation of the proximal tibia. |
Changes in the posteromedial proximal tibial epiphysis that lead to growth suppression. Any varus angulation at the knee in children > 2 y is considered abnormal. |
Pseudarthrosis of the tibia |
Anterolateral bowing. |
Almost always unilateral. Associated with neurofibromatosis (40%–50% of pseudarthroses of the tibia). |
Other syndromes and dysplasias
Fig. 5.93, p. 559
Fig. 5.94, p. 559
Fig. 5.95a, b, p. 559 |
|
Osteogenesis imperfecta, achondroplasia, and camp-omelic dysplasia, for example. |