KEY FACTS
Terminology
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Portal hypertension: Pressure gradient > 12 mm Hg between main portal vein (PV) and IVC &/or hepatic veins
Imaging
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Ultrasound for 1st-line evaluation of portal hypertension
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Best tool for dynamic evaluation of portal venous system
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Large main PV
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> 16 mm in maximal diameter
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Presence of portosystemic collateral vessels
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May also see slow flow in main PV, to-and-fro or bidirectional PV, or reversal of flow in PVs
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Coronary vein, a.k.a. left gastric vein, can become dilated in portal hypertension
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PVs including main portal, left portal, and anterior and posterior branches of right PV evaluated with both grayscale and color Doppler in longitudinal plane; spectral Doppler waveform analysis also performed on all vessels in longitudinal plane
Pathology
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Increased portal pressures causes portal blood flow to divert into portosystemic collaterals, eventually resulting in increased resistance and reversal of flow in PVs
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Intrahepatic etiologies most common cause of portal hypertension
Clinical Issues
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Patients often asymptomatic; clinical manifestations can include splenomegaly, abdominal wall collateral vessels, and thrombocytopenia
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Pathophysiology unclear, but thought to arise from hepatic inflammation with cellular regeneration, resulting in increased pressure in portal venous system
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Treatment of underlying cause of portal hypertension, if possible (i.e., infection)
Scanning Tips
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PV diameter on deep inspiration higher than on expiration; thus PV diameter measured during deep inspiration may overestimate true diameter